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压力导致50%衰老标识,积极上班反而更解压?比摸鱼更有效的富人解压方式
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[1] Biological age is increased by stress and restored upon recovery. [2] .Contextual modifiers of healthspan, lifespan, and epigenome in mice under chronic social stress. [3] Epigenetics as a key link between psychosocial stress and aging: concepts, evidence, mechanisms, [4 Stress-induced remodeling of lymphoid innervation. [5]Psychological stress and endocrine axes. Human Psychoneuroimmunology, [6] Glucocorticoids induce production of reactive oxygen species/reactive nitrogen species and DNA damage through an iNOS mediated pathway in breast cancer. [7] A stress response pathway regulates DNA damage through β2-adrenoreceptors and β-arrestin-1. [8] Good stress, bad stress and oxidative stress: Insights from anticipatory cortisol reactivity. [9] Four faces of cellular senescence. [10] Cellular senescence: when bad things happen to good cells. [11]Monoamine Oxidases, Oxidative Stress, and Altered Mitochondrial Dynamics in Cardiac Ageing. [12] Accelerated telomere shortening in response to life stress. [13] Prenatal maternal stress prospectively relates to shorter child buccal cell telomere length. [14]Influence of cortisol awakening response on telomere length: Trends for males and females. [15] Accelerated DNA methylation age in adolescent girls: associations with elevated diurnal cortisol and reduced hippocampal volume. [16] Epigenetic upregulation of FKBP5 by aging and stress contributes to NF-κB-driven inflammation and cardiovascular risk. Proceedings of the National Academy of [17] Mitochondria, telomeres and cell senescence. [18] Role of mitochondrial dysfunction and altered autophagy in cardiovascular aging and disease: from mechanisms to therapeutics. American journal of physiology. [19]Stress-induced biological aging: A review and guide for research priorities. [20] Chronic stress exposure and daily stress appraisals relate to biological aging marker p16INK4a.
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